A5049159118- Genetic factors in colorectal cancer
- High-Temperature Coating Behaviors
- Catalytic Processes in Materials Science
- Colorectal Cancer Screening and Detection
- T-cell and B-cell Immunology
- Nutrition, Genetics, and Disease
- Neuroinflammation and Neurodegeneration Mechanisms
- Gut microbiota and health
- Immune cells in cancer
- Thermochemical Biomass Conversion Processes
University of North Dakota
1986-2024
Colon cancer affects people of all ages. However, its frequency, as well the related morbidity and mortality, are high among older adults. The complex physiological changes in aging gut substantially limit development therapies. Here, we identify a unique intestinal microenvironment that is linked with an increased risk colon Our findings show markedly influences persistent fucosylation apical surfaces epithelial cells, which results favorable environment for tumor growth. Furthermore, our...
The low temperature oxidation of a Montana subbituminous coal was investigated using round bottom 100 ml flasks in constant baths. experiments were carried out normal and oxygen enriched air at 30°C, 45°C 70°C with particle sizes ranging from 4 mesh to mesh. Periodic analysis gas samples the provided rate data. reactivity as received compared that same dried (i) high pressure steam (ii) hot water A equation has been proposed incorporating effects diffusion surface reaction. For higher...
Background: Diet-induced obesity (DIO) increases risk for cardiovascular disease largely due to compromised gut barrier integrity and systemic inflammation. Animal models understanding the pathophysiology of complications associated with DIO do not take address genetic factors, which determine individual variation in susceptibility develop complications. Hypothesis: DO mice recapitulate heterogeneity western diet-induced metabolic syndrome, disruption Methods: were fed either control (CD) or...
Abstract A decline in mitochondrial activity has been linked to T cell dysfunction and the progression of a variety diseases. It is unclear whether function regulatory cells (Tregs) determines effector fate decisions regulates steady-state inflammation. We show that loss Mitochondrial Transcription Factor Foxp3 + Tregs (TFAM cKO) affects cellular transcriptional dynamics CD4 cells, resulting disproportional heterogeneity prevalence pathogenic subsets. Failure modulate responses results...