Belén Bellido

ORCID: 0000-0003-0464-6290
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About
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Research Areas
  • Chronic Kidney Disease and Diabetes
  • Autophagy in Disease and Therapy
  • Dialysis and Renal Disease Management
  • Organ Transplantation Techniques and Outcomes
  • Metabolism and Genetic Disorders
  • Cannabis and Cannabinoid Research
  • DNA Repair Mechanisms
  • Biomedical Research and Pathophysiology
  • Heme Oxygenase-1 and Carbon Monoxide
  • Coenzyme Q10 studies and effects
  • Free Radicals and Antioxidants
  • Endoplasmic Reticulum Stress and Disease
  • Liver Disease and Transplantation
  • Radiation Effects and Dosimetry
  • GABA and Rice Research
  • Birth, Development, and Health
  • Carcinogens and Genotoxicity Assessment

Universidad Nacional Autónoma de México
2017-2022

Endoplasmic reticulum (ER) stress is induced by intrinsic and extrinsic factors and, in consequence, activates the unfolded protein response (UPR); which an acute phase restores cellular homeostasis, whereas a chronic promotes apoptosis. ER has been detected broad range of kidney diseases different degrees UPR stimulation could activate antioxidant immune responses, autophagy, cell maintenance, inflammation, apoptosis fibrosis. Understanding regulation signaling pathways will allow to...

10.1016/j.arres.2021.100010 article EN cc-by-nc-nd Advances in Redox Research 2021-05-27

Kidney diseases encompass many pathologies, including obstructive nephropathy (ON), a common clinical condition caused by different etiologies such as urolithiasis, prostatic hyperplasia in males, tumors, congenital stenosis, and others. Unilateral ureteral obstruction (UUO) rodents is an experimental model widely used to explore the pathophysiology of ON, replicating vascular alterations, tubular atrophy, inflammation, fibrosis development. In addition, due kidney's high energetic demand,...

10.3390/metabo12100936 article EN cc-by Metabolites 2022-10-02

The raw data showed in this article comes from the published research entitled "Protective effects of Chlorogenic acid 3-Nitropropionic induced toxicity and genotoxicity" Food Chem Toxicol. 2017 May 3. pii: S0278-6915(17)30226-0. DOI:10.1016/j.fct.2017.04.048. [1]. Data illustrates antitoxic antigenotoxic (CGA) on genotoxicity produced by vivo treatment with mitochondria toxin (3-NP) mice. Toxicity was evaluated erythrocytes peripheral blood through micronuclei assay. share at Elsevier...

10.1016/j.dib.2017.07.004 article EN cc-by Data in Brief 2017-07-12
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