Desiree Williams

ORCID: 0009-0003-4944-848X
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About
Contact & Profiles
Research Areas
  • Coronary Interventions and Diagnostics
  • Antiplatelet Therapy and Cardiovascular Diseases
  • Atherosclerosis and Cardiovascular Diseases
  • Advanced Surface Polishing Techniques
  • Cell Adhesion Molecules Research
  • Biological Stains and Phytochemicals
  • Acute Myocardial Infarction Research
  • Immune cells in cancer

CVPath Institute
2024

BACKGROUND: Cell phenotype switching is increasingly being recognized in atherosclerosis. However, our understanding of the exact stimuli for such cellular transformations and their significance human atherosclerosis still evolving. Intraplaque hemorrhage thought to be a major contributor plaque progression part by stimulating influx CD163 + macrophages. Here, we explored hypothesis that macrophages cause through induction proapoptotic endothelial-to-mesenchymal transition (EndMT) within...

10.1161/circresaha.123.324082 article EN Circulation Research 2024-06-11
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