OsNBL3, a mitochondrion‐localized pentatricopeptide repeat protein, is involved in splicing nad5 intron 4 and its disruption causes lesion mimic phenotype with enhanced resistance to biotic and abiotic stresses

• DOI: 10.1111/pbi.13659
• Publication Date: 2021-07-02T07:36:57Z
• Authors: Tiancheng Qiu; Xiaosheng Zhao; Huijing Feng; Linlu Qi; Jun Yang; You‐Liang Peng; Wensheng Zhao
• Keywords: Pentatricopeptide repeat

Lesion mimic mutants are used to elucidate mechanisms controlling plant responses pathogen attacks and environmental stresses. Although dozens of genes had been functionally demonstrated be involved in lesion phenotype several species, the molecular underlying hypersensitive response largely unknown. Here, a rice (Oryza sativa) mutant natural blight leaf 3 (nbl3) was identified from T-DNA insertion lines. The causative gene, OsNBL3, encodes mitochondrion-localized pentatricopeptide repeat (PPR) protein. nbl3 exhibited spontaneous cell death H