About
Contact & Profiles
A5036622965
Research Areas
- Metabolism, Diabetes, and Cancer
- Amino Acid Enzymes and Metabolism
- CRISPR and Genetic Engineering
- Parkinson's Disease Mechanisms and Treatments
- Lysosomal Storage Disorders Research
- Autophagy in Disease and Therapy
- Animal Genetics and Reproduction
- Epigenetics and DNA Methylation
- Pancreatic function and diabetes
- Genetics and Neurodevelopmental Disorders
Helsinki University Hospital
2018
University of Helsinki
2016-2018
Mutations in PINK1 and Parkin result autosomal recessive Parkinson's disease (PD). Cell culture vitro studies have elaborated the PINK1-dependent regulation of defined how this dyad orchestrates elimination damaged mitochondria via mitophagy. phosphorylates ubiquitin at serine 65 (Ser65) an equivalent Ser65 residue located within its N-terminal ubiquitin-like domain, resulting activation; however, physiological significance phosphorylation vivo mammals remains unknown. To address this, we...
10.1098/rsob.180108
article
EN
cc-by
Open Biology
2018-11-01
10.1016/j.neurobiolaging.2016.10.014
article
EN
Neurobiology of Aging
2016-10-19
10.6084/m9.figshare.c.4269695.v2
article
EN
2018-11-08
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