A5076594108- Neuroinflammation and Neurodegeneration Mechanisms
- Venomous Animal Envenomation and Studies
- Stress Responses and Cortisol
- Tryptophan and brain disorders
- Adipose Tissue and Metabolism
- Social and Economic Solidarity
- Infant Nutrition and Health
- Child Nutrition and Feeding Issues
- International Relations and Autism
- Rabies epidemiology and control
- Neuroscience and Neuropharmacology Research
- Sirtuins and Resveratrol in Medicine
- Cellular transport and secretion
- Ion channel regulation and function
- Toxin Mechanisms and Immunotoxins
- Viral Infections and Vectors
- Autism Spectrum Disorder Research
- Vector-borne infectious diseases
- Genetic Neurodegenerative Diseases
- Biochemical and Structural Characterization
- Pancreatic function and diabetes
- Public Health in Brazil
Universidade Federal de Minas Gerais
2011-2023
Universidade Estadual de Santa Cruz
2019
Universidade Federal do Recôncavo da Bahia
2010
Obesity represents a global health problem and is characterized by metabolic dysfunctions low-grade chronic inflammatory state, which can increase the risk of comorbidities, such as atherosclerosis, diabetes insulin resistance. Here we tested hypothesis that genetic deletion metabotropic glutamate receptor 5 (mGluR5) may rescue features present in BACHD mice, mouse model Huntington's disease (HD) with an obese phenotype. For that, crossed mGluR5 knockout mice (mGluR5
A cross-sectional study was conducted to determine the seroprevalence of anti-Rickettsia spp. in dogs from Belo Horizonte, MG.For this purpose, serum samples 453 were collected during rabies vaccination campaign and tested by indirect immunofluorescence assay (IFA) using five antigens: Rickettsia bellii, amblyommii, rickettsii, parkeri, felis.The endpoint titer reacting with each antigen determined considered positive if it reacted at 1:64 dilution.Serum showing for a species least four-fold...
Chronic consumption of hyperpalatable and hypercaloric foods has been pointed out as a factor associated with cognitive decline memory impairment in obesity. In this context, the integration between peripheral central inflammation may play significant role negative effects an obesogenic environment on memory. However, little is known about how obesity-related affects specific neurotransmission systems involved regulation. Here, we test hypothesis that chronic exposure to highly palatable...
Abstract Chronic consumption of hyperpalatable and hypercaloric foods has been pointed out as a factor associated with cognitive decline memory impairment in obesity. In this context, the integration between peripheral central inflammation may play significant role negative effects an obesogenic environment on memory. However, little is known about how obesity-related affects specific neurotransmission systems involved regulation. Here, we test hypothesis that chronic exposure to highly...