Mahsa Noroozzadeh

ORCID: 0000-0003-4561-8472
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About
Contact & Profiles
Research Areas
  • Ovarian function and disorders
  • Reproductive Biology and Fertility
  • Birth, Development, and Health
  • Hormonal and reproductive studies
  • Pregnancy and preeclampsia studies
  • Gestational Diabetes Research and Management
  • Menopause: Health Impacts and Treatments
  • Growth Hormone and Insulin-like Growth Factors
  • Reproductive System and Pregnancy
  • Endometriosis Research and Treatment
  • Sexual Differentiation and Disorders
  • Phytoestrogen effects and research
  • Sexual function and dysfunction studies
  • Blood Pressure and Hypertension Studies
  • Digestive system and related health
  • Pregnancy-related medical research
  • Ovarian cancer diagnosis and treatment
  • Reproductive Health and Technologies
  • Lipid metabolism and disorders
  • Demographic Trends and Gender Preferences
  • Sperm and Testicular Function
  • Reproductive Health and Contraception
  • Thyroid Disorders and Treatments
  • GABA and Rice Research
  • Neuroendocrine regulation and behavior

Research Institute for Endocrine Sciences
2016-2025

Shahid Beheshti University of Medical Sciences
2016-2025

Denver School of Nursing
2022

Triangle
2022

Indianapolis Zoo
2022

Gainesville Obstetrics & Gynecology
2022

Committee on Publication Ethics
2022

Endocrinology Research Center
2014

Islamic Azad University North Tehran Branch
2012

Polycystic ovary syndrome (PCOS) is one of the most common endocrine disorders in women, with a prevalence 8-12% during reproductive years. In present study, using prenatal exposure to single dose testosterone critical period fetal development, we aimed introduce an enhanced rat model that would exhibit both and ovarian disturbances similar PCOS, while maintaining normal system morphology adulthood. Ten pregnant rats were injected s.c. 5 mg free on gestational day 20, control received only...

10.1113/expphysiol.2014.078055 article EN Experimental Physiology 2014-02-15

Polycystic ovary syndrome (PCOS) is one of the most common reproductive disorders in women. Previous studies have shown that prenatal exposure female fetuses to androgen can be considered an important factor development PCOS.In present study we aimed examine effects rat previously documented doses testosterone on different embryonic days PCOS phenotype adulthood.Pregnant rats were divided into four groups, experimental and control groups. Three mg free was administered subcutaneously group 1...

10.5812/ijem.16502 article EN cc-by-nc International Journal of Endocrinology and Metabolism 2014-04-01

Genetic factors and an unfavorable intrauterine environment may contribute to the development of metabolic disorders in offspring later life. The present study aims investigate compare risks pre-diabetes mellitus (pre-DM), type 2 diabetes (T2DM) abnormal glucose tolerance female male with early maternal menopausal age versus those normal age, In this prospective population-based study, there were 1,516 females 1,563 males as well 213 237 age. Unadjusted adjusted cox regression models used...

10.1186/s12958-025-01405-z article EN cc-by-nc-nd Reproductive Biology and Endocrinology 2025-05-22

The reproductive system is extremely susceptible to environmental insults, for example exogenous steroids during gestational development and differentiation. Experimental induction of androgen excess prenatal life in female animal models reprograms their physiology, however the fetal programming male by has not been well studied. We aimed determine effect exposure two different doses testosterone on days, using a rat model. Sixteen pregnant rats were randomly divided into experimental groups...

10.1371/journal.pone.0071705 article EN cc-by PLoS ONE 2013-08-15

10.1016/j.ejogrb.2013.09.029 article EN European Journal of Obstetrics & Gynecology and Reproductive Biology 2013-10-18

Abstract Aims Impaired implantation due to the reduced endometrial receptivity considers an etiology for infertility in polycystic ovary syndrome (PCOS). In this context, we aimed compare expression of interleukin 10 ( Il10 ), homeobox A10 Hoxa10 signal transducer and activator transcription 3 Stat3 β3‐integrin Itgb3 ) embryo site a prenatally‐androgenized rat model PCOS before during gestation. Materials Methods was created by injection testosterone prenatally. The uterine tissues were...

10.1111/aji.13702 article EN American Journal of Reproductive Immunology 2023-04-16

The impact of endogenous estrogen exposure (EEE) on hypertension (HTN) incidence has not been investigated yet.

10.1210/clinem/dgae316 article EN publisher-specific-oa The Journal of Clinical Endocrinology & Metabolism 2024-05-09
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