Nahid Akhtar

ORCID: 0009-0003-8816-4690
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About
Contact & Profiles
Research Areas
  • Dermatology and Skin Diseases
  • Allergic Rhinitis and Sensitization
  • Exercise and Physiological Responses
  • Immune Cell Function and Interaction
  • Toxin Mechanisms and Immunotoxins
  • PARP inhibition in cancer therapy
  • Reproductive System and Pregnancy
  • Ion Channels and Receptors

Indiana University – Purdue University Indianapolis
2012-2024

Indiana University School of Medicine
2012

Atopic dermatitis results in diminished barrier function and altered production of antimicrobial peptides. Dendritic epidermal T cells (DETCs) play an important role the wound repair inflammation process. Our previous work identified IL-4-dependent loss DETCs Stat6VT mice MC903-induced skin mouse model. However, mechanisms through which IL-4 mediates are unclear. In this study, we show that IL-4Rα germline knockout (Il4ra-/-) have increased DETCs, faster healing, differentiation complex gene...

10.4049/jimmunol.2300629 article EN The Journal of Immunology 2024-05-24
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