Mengchuan Xu

ORCID: 0000-0002-3032-0166
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About
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Research Areas
  • Cellular and Composite Structures
  • Mechanical Behavior of Composites
  • Thermal properties of materials
  • Polymer composites and self-healing
  • Autophagy in Disease and Therapy
  • Automotive and Human Injury Biomechanics
  • TGF-β signaling in diseases
  • PI3K/AKT/mTOR signaling in cancer
  • Advanced Materials and Mechanics
  • Structural Analysis of Composite Materials
  • Galectins and Cancer Biology
  • Protein Tyrosine Phosphatases
  • Radioactive element chemistry and processing
  • Structural Behavior of Reinforced Concrete
  • Genomics, phytochemicals, and oxidative stress
  • Nuclear materials and radiation effects
  • Thermal Expansion and Ionic Conductivity
  • Structural Response to Dynamic Loads
  • Glioma Diagnosis and Treatment
  • Glycosylation and Glycoproteins Research
  • Hedgehog Signaling Pathway Studies
  • Polymer Foaming and Composites
  • Additive Manufacturing and 3D Printing Technologies
  • Electrospun Nanofibers in Biomedical Applications
  • Transportation Safety and Impact Analysis

Ministry of Industry and Information Technology
2023

Beijing Institute of Technology
2019-2023

China Academy of Space Technology
2023

Corrugated sandwich panels with outstanding load-bearing and energy absorption capacities are widely used in aerospace, naval automobile industries. Herein, a series of modified sinusoidal corrugated (MSC) panels, multiple layers gradient design, developed fabricated by additive manufacturing. An analytical model for quasi-static compression performance was established using Timoshenko beam plastic hinge theory. Corresponding experiments finite element analysis conducted to investigate the...

10.1016/j.matdes.2019.107858 article EN cc-by-nc-nd Materials & Design 2019-05-17

10.1016/j.ijsolstr.2022.111866 article EN publisher-specific-oa International Journal of Solids and Structures 2022-07-16

Cancer cells consistently utilize the unfolded protein response (UPR) to encounter abnormal endoplasmic reticulum (ER) stress induced by accumulation of misfolded proteins. Extreme activation UPR could also provoke maladaptive cell death. Previous reports have shown that NRF2 antioxidant signaling is activated and serves as noncanonical pathway defense reduce excessive ROS levels during ER stress. However, mechanisms regulating upon in glioblastoma not been fully elucidated. Here we identify...

10.1038/s41419-023-05873-2 article EN cc-by Cell Death and Disease 2023-06-14
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