- Neurological disorders and treatments
- Neurotransmitter Receptor Influence on Behavior
- Parkinson's Disease Mechanisms and Treatments
- Neuroscience and Neuropharmacology Research
- Neuroscience of respiration and sleep
- Winter Sports Injuries and Performance
- Cryospheric studies and observations
- Neurogenesis and neuroplasticity mechanisms
- Cholinesterase and Neurodegenerative Diseases
- Mitochondrial Function and Pathology
- Sports injuries and prevention
- Photonic Crystals and Applications
- Glycogen Storage Diseases and Myoclonus
- Quantum optics and atomic interactions
- Neurological diseases and metabolism
- Neuropeptides and Animal Physiology
- Diet and metabolism studies
- Regulation of Appetite and Obesity
- Receptor Mechanisms and Signaling
- Genetic Neurodegenerative Diseases
- Ion channel regulation and function
- Vestibular and auditory disorders
- Ophthalmology and Eye Disorders
- Botulinum Toxin and Related Neurological Disorders
- Adipose Tissue and Metabolism
National Research Council Canada
2014-2025
University of Calgary
1981-2011
Université de Moncton
2001-2002
Hôpital Maisonneuve-Rosemont
1999
Université de Montréal
1955-1999
McGill University
1960-1995
Jewish General Hospital
1995
Hôpital de l'Enfant-Jésus
1977-1988
Deleted Institution
1981
Royal College of Physicians and Surgeons of Canada
1981
Journal Article INFLUENCE OF THE SUBSTANTIA NIGRA ON CATECHOLAMINE CONTENT STRIATUM Get access L. J. POIRIER, POIRIER Search for other works by this author on: Oxford Academic PubMed Google Scholar T. SOURKES Brain, Volume 88, Issue 1, March 1965, Pages 181–192, https://doi.org/10.1093/brain/88.1.181 Published: 01 1965
Journal Article STRIATAL AMINES, EXPERIMENTAL TREMOR AND THE EFFECT OF HARMALINE IN MONKEY Get access L. J. POIRIER, POIRIER Laboratoire des Sciences Neurologiques, Faculté de Médecine, Université MontréalAllan Memorial Institute of Psychiatry, McGill University Search for other works by this author on: Oxford Academic PubMed Google Scholar T. SOURKES, SOURKES G. BOUVIER, BOUVIER R. BOUCHER, BOUCHER S. CARABIN Brain, Volume 89, Issue 1, March 1966, Pages 37–52,...
IN previous studies in the monkey, it was observed that there is a direct relationship betwen interruption of ascending nervous fibers at level ventromedial area upper brain stem, retrograde cellular degeneration ipsilateral substantia nigra,<sup>1</sup>and depletion dopamine both parts homolateral striatum.<sup>2-4</sup>Similarly, serotonin striatum attributed to most dorsomedial cerebral peduncle on corresponding side.<sup>5,6</sup>These neuroanatomical and neurochemical changes are...
Abstract A morphological study of the red nucleus and its efferent nervous pathways based on retrograde cellular degeneration (cell loss) degenerated fibers impregnated with osmic acid gave folowing results. Interruption rubro‐tegmentospinal tract was associated a complete disappearance cells in contralateral magnocellular some cell loss adjoining tegmental gray. central bundle results parvicellular part ipsilateral nucleus. Such lesion is rubro‐olivary that terminate along dorsal lamella...
Lesions were placed in the left ventromedial tegmental area of brains nine monkeys (Macaca mulatto). After 1–4 months, removed, and striatum was dissected prepared for chemical analysis. In seven animals which lesion strictly unilateral, striatal dopamine very much reduced below level found on intact side brain. two impinged right brain, concentration both sides. Homovanillic acid 3,4-dihydroxyphenylacetic acid, metabolic derivatives dopamine, measured striata four five respectively. Both...
The spontaneous activily of the putamen and related structures was studied on both sides brain in cats with chronic unilateral lesions interrupting corresponding nigrostriatal pathway at level upper midbrain or area medial forebrain bundle. frequency activity recorded these animals after high cervical cord transection greater deprived its incoming fibers than intact side normal cat. Under such conditions l-dopa increased lesion but not side. These results suggest that absence endings...