Quentin Bruggeman

ORCID: 0000-0002-2288-7744
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About
Contact & Profiles
Research Areas
  • Photosynthetic Processes and Mechanisms
  • Plant Stress Responses and Tolerance
  • Plant Molecular Biology Research
  • Plant-Microbe Interactions and Immunity
  • Plant Gene Expression Analysis
  • Plant Disease Resistance and Genetics
  • Chromosomal and Genetic Variations
  • DNA Repair Mechanisms
  • Plant Reproductive Biology
  • Plant responses to water stress
  • Plant nutrient uptake and metabolism
  • Plant Genetic and Mutation Studies
  • Genomics and Chromatin Dynamics
  • Mitochondrial Function and Pathology

Institut des Sciences des Plantes de Paris Saclay
2014-2020

Université Paris Cité
2016

Centre National de la Recherche Scientifique
2012-2016

Sorbonne Paris Cité
2016

Université Paris-Sud
2012-2016

Institut de Biologie Moléculaire des Plantes
2014

Abstract Programmed cell death (PCD) is essential for several aspects of plant life, including development and stress responses. Indeed, incompatible plant-pathogen interactions are well known to induce the hypersensitive response, a localized death. Mutational analyses have identified key PCD components, we recently mips1 mutant Arabidopsis (Arabidopsis thaliana), which deficient enzyme catalyzing limiting step myoinositol synthesis. One most striking features light-dependent formation...

10.1104/pp.114.236083 article EN PLANT PHYSIOLOGY 2014-04-04

Abstract Programmed cell death (PCD) is essential for several aspects of plant life, including development and stress responses. We recently identified the mips1 mutant Arabidopsis thaliana, which deficient enzyme catalyzing limiting step myo-inositol (MI) synthesis. One most striking features light-dependent formation lesions on leaves due to salicylic acid (SA)-dependent PCD. Here, we a suppressor PCD by screening mutations that abolish phenotype. Our screen hxk1 mutant, mutated in gene...

10.1105/tpc.15.00068 article EN The Plant Cell 2015-06-01

Abstract Programmed cell death (PCD) is essential for several aspects of plant life. We previously identified the mips1 mutant Arabidopsis thaliana , which deficient enzyme catalysing myo-inositol synthesis, and that displays light-dependent formation lesions on leaves due to Salicylic Acid (SA) over-accumulation. Rationale this work was identify novel regulators PCD using a genetic approach. A screen secondary mutations abolish phenotype mutation in BIG gene, encoding factor unknown...

10.1038/s41598-020-68235-4 article EN cc-by Scientific Reports 2020-07-09

Programmed cell death (PCD) is a crucial process both for plant development and responses to biotic abiotic stress. There accumulating evidence that chloroplasts may play central role during PCD as mitochondria in animal cells, but it still unclear whether they participate onset, execution, or both. To tackle this question, we have analyzed the contribution of chloroplast function phenotype myoinositol phosphate synthase1 (mips1) mutant forms spontaneous lesions light-dependent manner. We...

10.1104/pp.15.01872 article EN PLANT PHYSIOLOGY 2016-01-08
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