About
Contact & Profiles
A5061145894
Research Areas
- Alzheimer's disease research and treatments
- Neuroscience and Neuropharmacology Research
- Cholinesterase and Neurodegenerative Diseases
- Hormonal and reproductive studies
- Functional Brain Connectivity Studies
- Hormonal Regulation and Hypertension
- Genetic Syndromes and Imprinting
- Memory and Neural Mechanisms
- Neurological diseases and metabolism
- Amino Acid Enzymes and Metabolism
- Neuroinflammation and Neurodegeneration Mechanisms
- Inflammatory mediators and NSAID effects
- Folate and B Vitamins Research
- Parkinson's Disease Mechanisms and Treatments
University of Padua
2006-2020
Consejo Nacional de Investigaciones Científicas y Técnicas
1979
10.1016/j.neurobiolaging.2014.10.020
article
EN
Neurobiology of Aging
2014-10-20
10.1016/j.neurobiolaging.2016.10.027
article
EN
Neurobiology of Aging
2016-11-05
Senile plaques, the hallmarks of Alzheimer’s Disease (AD), are generated by deposition amyloid-beta (Aβ), proteolytic product amyloid precursor protein (APP), β and γ-secretase. A large body evidence points towards a role for Ca2+ imbalances in pathophysiology both sporadic familial forms AD (FAD). reduction store-operated entry (SOCE) is shared numerous FAD-linked mutations, SOCE involved Aβ accumulation different model cells. In neurons, components remain quite obscure, whereas astrocytes,...
10.3390/ijms21155288
article
EN
International Journal of Molecular Sciences
2020-07-25
10.1016/0090-6980(79)90120-5
article
EN
Prostaglandins
1979-12-01
10.1177/172460080602100209
article
The International Journal of Biological Markers
2006-04-01
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