Daniel Boland

ORCID: 0000-0003-2887-8117
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About
Contact & Profiles
Research Areas
  • Protein Kinase Regulation and GTPase Signaling
  • Amyloidosis: Diagnosis, Treatment, Outcomes
  • Thyroid Disorders and Treatments
  • Monoclonal and Polyclonal Antibodies Research

Scripps Research Institute
2017

Increasing evidence supports the hypothesis that soluble misfolded protein assemblies contribute to degeneration of postmitotic tissue in amyloid diseases. However, there is a dearth reliable nonantibody-based probes for selectively detecting oligomeric aggregate structures circulating plasma or deposited tissues, making it difficult scrutinize this patients. Hence, understanding structure-proteotoxicity relationships driving diseases remains challenging, hampering development early...

10.1126/scitranslmed.aam7621 article EN Science Translational Medicine 2017-09-13

Searchable abstracts of presentations at key conferences in endocrinology ISSN 1470-3947 (print) | 1479-6848 (online)

10.1530/endoabs.41.gp190 article EN Endocrine Abstracts 2016-05-13
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