T. Miyake

ORCID: 0000-0003-3122-4528
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About
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Research Areas
  • Neurobiology and Insect Physiology Research
  • Dermatology and Skin Diseases
  • Cancer Cells and Metastasis
  • Gastrointestinal motility and disorders
  • Insect and Pesticide Research
  • Immunotherapy and Immune Responses
  • Autoimmune Bullous Skin Diseases
  • Viral Infectious Diseases and Gene Expression in Insects
  • Bacterial Genetics and Biotechnology
  • Invertebrate Immune Response Mechanisms
  • Asthma and respiratory diseases
  • Insect and Arachnid Ecology and Behavior
  • Immunodeficiency and Autoimmune Disorders
  • RNA and protein synthesis mechanisms
  • Developmental Biology and Gene Regulation
  • T-cell and B-cell Immunology
  • Insect symbiosis and bacterial influences
  • Chronic Lymphocytic Leukemia Research
  • Bacteriophages and microbial interactions
  • Psoriasis: Treatment and Pathogenesis
  • Vascular Malformations and Hemangiomas
  • Plant Toxicity and Pharmacological Properties
  • Wnt/β-catenin signaling in development and cancer
  • Chemical Reactions and Isotopes
  • Organ and Tissue Transplantation Research

Kyoto University
2019-2024

Tenri Hospital
2015

Mitsubishi Group (Japan)
1977-1996

Fujirebio (Japan)
1996

Kitasato University
1990

Institute of Life Sciences
1989

Keio University
1971

Aichi Cancer Center
1970

Kyoto University Hospital
1967

In Drosophila sensory organ development, the balance of activities between proneural genes and repressor defines a cluster as population competent cells for neural development. this study, we report isolation analysis tamou (tam) gene that encodes cell-cell junction-associated protein, which is homologous to mammalian ZO-1, member membrane-associated guanylate kinase homolog family. The tam mutation reduces transcription gene, extramacrochaetae, causes enlargement where supernumerary...

10.1101/gad.10.14.1783 article EN Genes & Development 1996-07-15

We attempted to apply a new criterion, the template specificity of RNA replicases Escherichia coli phages, for grouping these phages. Based on specificity, it was shown that (a) Qbeta, VK, and ST belonged one group (group III), SP FI another IV), (b) some similarity existed between groups III IV. Considering such in addition previously reported properties phage particles, we could separate phages into four major (I-IV) with subgroups a, b, c III, b

10.1073/pnas.68.9.2022 article EN Proceedings of the National Academy of Sciences 1971-09-01

10.1007/bf02620984 article EN In Vitro Cellular & Developmental Biology 1987-10-01

Syringocystadenocarcinoma papilliferum (SCACP) is a very rare cutaneous adnexal neoplasm. SCACP presents histologic variability, and it difficult to establish the diagnosis from punch biopsy. has an overall configuration similar that of syringocystadenoma (SCAP). When we diagnose SCACP, features SCAP can be contributing immunohistochemical staining useful. Our case shows variability pitfalls biopsy for SCACP.

10.1159/000381940 article EN cc-by-nc Case Reports in Dermatology 2015-05-07

Abstract Background The circadian rhythm controls multiple biological processes, including immune responses; however, its impact on cutaneous adaptive response remains unclear. Methods We used a well‐established type IV allergy model, contact hypersensitivity (CHS). induced CHS using dinitrofluorobenzene (DNFB). Mice were sensitized and elicited with DNFB in the daytime or at night. Results In mice, nocturnally active animal, we found that ear swelling increased when mice night compared...

10.1111/all.15314 article EN Allergy 2022-04-15

New RNA replicases were isolated and purified from Escherichia coli Q13 infected with SP or FI phages showing different serological properties. These showed a template specificity that of Qbeta replicase.

10.1073/pnas.68.8.1778 article EN Proceedings of the National Academy of Sciences 1971-08-01

After terminating the present series of experiments, we found in literature that Heston had employed analogous methodological approach order to confirm difference degree genetic susceptibility tumor formation susceptible strain A and resistant C57L lung shown factor development pulmonary mice resides per se (5).

10.3181/00379727-134-34822 article EN Experimental Biology and Medicine 1970-06-01
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