Marcus Mahar

ORCID: 0000-0003-3243-5052
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About
Contact & Profiles
Research Areas
  • Nerve injury and regeneration
  • Signaling Pathways in Disease
  • Neurogenesis and neuroplasticity mechanisms
  • Pancreatic function and diabetes
  • Supramolecular Self-Assembly in Materials
  • Hippo pathway signaling and YAP/TAZ
  • Microtubule and mitosis dynamics
  • Histone Deacetylase Inhibitors Research

Washington University in St. Louis
2018-2020

Hope Center for Neurological Disorders
2018

Significance Functional impairment and poor recovery following central nervous system injury is due to the failure of injured axons regenerate rebuild functional connections. In contrast, axon regeneration occurs in peripheral nerves. Expression regeneration-associated genes essential activate program. Less known about contribution gene inactivation. We reveal an epigenetic mechanism that silences expression by ubiquitin-like containing PHD ring finger 1 (UHRF1)-dependent DNA methylation...

10.1073/pnas.1812518115 article EN Proceedings of the National Academy of Sciences 2018-12-10
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