A5023576308- Cancer Genomics and Diagnostics
- Acute Lymphoblastic Leukemia research
- Chronic Lymphocytic Leukemia Research
- Musculoskeletal pain and rehabilitation
- Chronic Myeloid Leukemia Treatments
- Lung Cancer Treatments and Mutations
- RNA regulation and disease
- Melanoma and MAPK Pathways
- Folate and B Vitamins Research
- Brain Tumor Detection and Classification
- Motor Control and Adaptation
- Myofascial pain diagnosis and treatment
- melanin and skin pigmentation
- Mycobacterium research and diagnosis
- Mast cells and histamine
- Renal and related cancers
- Renal cell carcinoma treatment
- Head and Neck Cancer Studies
- Fungal Infections and Studies
- Orthopedic Surgery and Rehabilitation
- RNA modifications and cancer
- Neutropenia and Cancer Infections
- Sport Psychology and Performance
- Viral-associated cancers and disorders
- Advanced Breast Cancer Therapies
Division of Cancer Epidemiology and Genetics
2021
National Cancer Institute
2019-2021
National Institutes of Health
2021
Universidad de Guadalajara
2020
Instituto Nacional de Cancerología
2018
National Institute of Quality
2006
Abstract Epidemiologic studies often rely on questionnaire data, exposure measurement tools, and/or biomarkers to identify risk factors and the underlying carcinogenic processes. An emerging promising complementary approach investigate cancer etiology is study of somatic “mutational signatures” that endogenous exogenous processes imprint cellular genome. These signatures can be identified from a complex web mutations thanks advances in DNA sequencing technology analytical algorithms. This at...
To evaluate the effects of practice variability on chiropractic students' capacity to deliver spinal manipulations (SMs) a targeted peak force.Forty students participated in an experimental session including either variable or constant protocol 45 SMs. SMs were delivered computer-connected device that recorded force-time profiles. Ten with target force 350-N performed before practice, immediately following and 2 days later. Mixed-design analyses variance used assess effect type SM...
Abstract While several high-penetrance melanoma risk genes are known, variation in these fail to explain susceptibility a large proportion of high-risk families. As part family sequencing study, including 435 families from Mediterranean populations, we identified novel NRAS variant (c.170A>C, p.D57A) melanoma-prone family. This is absent exomes gnomAD, ESP, UKBiobank, and the 1000 Genomes Project, as well 11 273 individuals 109 US Australia. occurs GTP-binding pocket NRAS. Differently...