Marta Nadal

ORCID: 0009-0006-1776-0884
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About
Contact & Profiles
Research Areas
  • Medieval European Literature and History
  • Medieval Iberian Studies
  • Comparative Literary Analysis and Criticism
  • Archaeological and Historical Studies
  • Spanish Culture and Identity
  • Historical and Modern Theater Studies
  • Galician and Iberian cultural studies
  • Social Sciences and Policies
  • Spanish Literature and Culture Studies
  • Media, Journalism, and Communication History
  • Cultural and Mythological Studies
  • Spanish History and Politics
  • Libraries, Manuscripts, and Books
  • Historical and socio-economic studies of Spain and related regions
  • Medieval Architecture and Archaeology
  • Archaeological and Geological Studies
  • History, Culture, and Society
  • Cytomegalovirus and herpesvirus research
  • Neurogenetic and Muscular Disorders Research
  • Legionella and Acanthamoeba research
  • Nationalism and Cultural Identity
  • Classical Studies and Legal History
  • Cinema History and Criticism
  • Palliative and Oncologic Care
  • Aging, Health, and Disability

Hospital de Sant Pau
2022

Bacteria often encounter physico-chemical stresses that disrupt division, leading to filamentation, where cells elongate without dividing. While this adaptive response enhances survival, it also exposes filaments significant mechanical strain, raising questions about the mechanochemical feedback in bacterial systems. In study, we investigate how strain influences Min oscillatory system, a reaction-diffusion network central division Escherichia coli . Through multidisciplinary approach...

10.1101/2025.02.21.638242 preprint EN cc-by bioRxiv (Cold Spring Harbor Laboratory) 2025-02-26

Spinal muscular atrophy (SMA) is a neurodegenerative disease caused by the absence of functional copy Survival Motor Neuron 1 gene (SMN1). The nearly identical paralog, SMN2, cannot compensate for loss SMN1 because exon 7 aberrantly skipped from most SMN2 transcripts, process mediated synergistic activities Src-associated during mitosis, 68 kDa (Sam68/KHDRBS1) and heterogeneous nuclear ribonucleoprotein (hnRNP) A1. This results in production truncated, nonfunctional protein that rapidly...

10.1002/pro.4553 article EN Protein Science 2022-12-23
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