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K Kasliwal

ORCID: 0000-0003-0004-1593
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About
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A5115575515
Research Areas
  • RNA modifications and cancer
  • Epigenetics and DNA Methylation
  • Pluripotent Stem Cells Research
  • Genomics and Chromatin Dynamics
  • Cancer-related molecular mechanisms research
  • Genetic Syndromes and Imprinting

Cornell University
 2024-2025

Weill Cornell Medicine
 2024

 Genetic variation modulates susceptibility to aberrant DNA hypomethylation and imprint deregulation in naive pluripotent stem cells
OPENALEX - Publications 
Chirag R. Parikh Rachel A. Glenn Yufang Shi K Chatterjee K Kasliwal and 12 more Emily Swanzey Samuel Singer Su-Il Do Yingqian A. Zhan Yasuhide Furuta Mamta Tahiliani Eirini Apostolou A. Polyzos Richard P. Koche Jason G. Mezey Thomas Vierbuchen Matthias Stadtfeld

10.1016/j.stemcr.2025.102450 article EN cc-by-nc-nd Stem Cell Reports 2025-03-01
 Coordinated repression of totipotency-associated gene loci by histone methyltransferase EHMT2 through binding to LINE-1 regulatory elements
OPENALEX - Publications 
Kaushiki Chatterjee Christopher M. Uyehara K Kasliwal Subhashini Madhuranath Laurianne Scourzic and 3 more A. Polyzos Effie Apostolou Matthias Stadtfeld

SUMMARY Mouse embryonic stem cells (mESCs) and other naïve pluripotent can reverse typical developmental trajectories and, at low frequency, de-differentiate into 2-cell-like (2CLCs) that resemble the mammalian embryo during zygotic genome activation (ZGA). This affords opportunity to reveal molecular principles govern pre-implantation stages of development. We leveraged a multipurpose allele for acute protein depletion efficient immunoprecipitation dissect functions chromatin repressor...

10.1101/2024.12.18.629181 preprint EN cc-by-nd bioRxiv (Cold Spring Harbor Laboratory) 2024-12-20
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