A5046077915- Urinary Bladder and Prostate Research
- Advanced Glycation End Products research
- Cancer, Hypoxia, and Metabolism
- Aldose Reductase and Taurine
- RNA modifications and cancer
- Diabetes Management and Research
- Cancer-related Molecular Pathways
- Liver Disease Diagnosis and Treatment
- Urinary Tract Infections Management
- Endometriosis Research and Treatment
- Plant Toxicity and Pharmacological Properties
- Tumors and Oncological Cases
- Thermal Regulation in Medicine
- Cancer Diagnosis and Treatment
- Ubiquitin and proteasome pathways
- Endoplasmic Reticulum Stress and Disease
- Diabetes and associated disorders
- Pelvic floor disorders treatments
- Ferroptosis and cancer prognosis
- Diet, Metabolism, and Disease
- Microtubule and mitosis dynamics
- Sepsis Diagnosis and Treatment
- Cancer-related gene regulation
- Alcohol Consumption and Health Effects
- Inflammasome and immune disorders
China Medical University
2014-2021
First Hospital of China Medical University
2014-2021
Case Western Reserve University
2012-2016
MetroHealth Medical Center
2016
University Hospitals Cleveland Medical Center
2012-2015
Diabetes mellitus causes diabetic bladder dysfunction. We identified the pathogenic roles of polyuria and hyperglycemia in dysfunction rats.A total 72 female Sprague-Dawley® rats were divided into 6 groups, including age matched controls, with sham urinary diversion, streptozotocin induced diabetes after diversion 5% sucrose diuresis diversion. Urinary was performed by ureterovaginostomy 10 days before induction. Animals evaluated 20 weeks or measured 24-hour drinking voiding volumes,...
Objectives To determine whether diabetes mellitus‐ and diuresis‐induced alterations in the bladder can be reversed rats. Methods Male Sprague–Dawley rats were randomly distributed into eight groups ( n = 16 per group): 3 weeks 11 age‐matched controls, after streptozotocin‐induced mellitus, mellitus induction then treated with insulin for 8 weeks, 5% sucrose‐induced diuresis, diuresis followed by removal of sucrose weeks. Bodyweight, blood glucose glycated hemoglobin A1c monitored. At...
Manganese superoxide dismutase (MnSOD) is considered a critical component of the antioxidant systems that protect against oxidative damage. We are interested in role stress bladder detrusor smooth muscle (SM) different disease states. In this study, we generated an inducible, SM-specific Sod2(-/-) mouse model to investigate effects MnSOD depletion on function bladder. crossbred floxed Sod2 (Sod2(lox/lox)) mice with containing heterozygous knock-in gene encoding tamoxifen-activated Cre...
Abstract The intracellular domain of UNC5B contains both death and caspase‐3 cleavage site, is regarded as a functional that mediates apoptosis. However, in our previous studies, we found the bladder cancer cells could not be activated to promote In this study, different truncates (residue 399‐945, residue 412‐945) were created explore whether site (site 412), another potential its portion, induce apoptosis cells. Using mass spectrometry, acquired comprehensive detailed identification...
You have accessJournal of UrologyBladder and Urethra: Anatomy, Physiology Pharmacology II1 Apr 2015MP21-18 CONDITIONAL DELETION OF MANGANESE SUPEROXIDE DISMUTASE IN DETRUSOR SMOOTH MUSCLE IS ASSOCIATED WITH BLADDER OVERACTIVITY MICE Rania Elrashidy, Guiming Liu, Nan Xiao, Michael Kavran, Yexiang Huang, Mingfang Tao, Hoda Mohammad, C Thomas Powell, Firouz Daneshgari ElrashidyRania Elrashidy More articles by this author , LiuGuiming Liu XiaoNan Xiao KavranMichael Kavran HuangYexiang Huang...