A5044824425- Asthma and respiratory diseases
- Pediatric health and respiratory diseases
- Allergic Rhinitis and Sensitization
- Immune Response and Inflammation
- Tannin, Tannase and Anticancer Activities
- Advanced Nanomaterials in Catalysis
- interferon and immune responses
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms
- IL-33, ST2, and ILC Pathways
- Food Allergy and Anaphylaxis Research
- Inflammasome and immune disorders
- Curcumin's Biomedical Applications
Baylor College of Medicine
2024
Background : IL4, IL5, IL13, and IL17-producing CD4 T helper 2 (Th2)-cells 17 (Th17)-cells contribute to chronic eosinophilic neutrophilic airway inflammation in asthma allergic inflammation. Chemokines their receptors are upregulated Th2-Th17-mediated However, the ability of CXCR1 CXCR2 inhibition suppress Th2 Th17 cell-mediated lung has not been reported. Methods Mice sensitized challenged with cat dander extract (CDE) mount a vigorous Ladarixin is an orally bioavailable allosteric...
Abstract This study investigated the fabrication of gallic acid‐loaded chitosan nanoparticles (Gal‐Chi‐NPs) that enhanced DNA damage and apoptotic features by inhibiting FEN‐1 expressions in MDA‐MB 231 cells. Gal‐Chi‐NPs were fabricated ionic gelation method, it was characterized several studies such as dynamic light spectroscopy, Fourier‐transforms infrared x‐ray diffraction, scanning electron microscopy, energy‐dispersive x‐ray, atomic force thermogravimetric analysis. We have obtained...
IL4, IL5, IL13, and IL17-producing CD4 T helper 2 (Th2)-cells 17 (Th17)-cells contribute to chronic eosinophilic neutrophilic airway inflammation in asthma allergic inflammation. Chemokines their receptors are upregulated Th2/Th17-mediated However, the ability of CXCR1 CXCR2 modulate Th2 Th17-cell-mediated lung has not been reported.