Elba Medina

ORCID: 0000-0002-6202-9939
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About
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Research Areas
  • COVID-19 Clinical Research Studies
  • Long-Term Effects of COVID-19
  • Inflammasome and immune disorders
  • SARS-CoV-2 and COVID-19 Research
  • Diabetes and associated disorders
  • Obesity and Health Practices
  • Biomedical Ethics and Regulation
  • Neonatal Health and Biochemistry
  • Sepsis Diagnosis and Treatment
  • Dialysis and Renal Disease Management
  • Ethics and bioethics in healthcare
  • Systemic Sclerosis and Related Diseases
  • Ion channel regulation and function
  • Vitamin D Research Studies
  • Parathyroid Disorders and Treatments
  • Renal Diseases and Glomerulopathies
  • Nutrition and Health in Aging
  • Pharmacological Effects and Toxicity Studies
  • Skin Diseases and Diabetes
  • Ion Transport and Channel Regulation
  • Renal function and acid-base balance
  • Pregnancy and preeclampsia studies

Hattiesburg Clinic
2025

Hospital General de México
2020-2025

Universidad Nacional Autónoma de México
2023-2024

Hospital Infantil de México Federico Gómez
2012

<ns3:p>Background Coronavirus disease 2019 (COVID-19) has created the need to evaluate drugs such as favipiravir (FPV), an antiviral inhibitor of RNA-dependent RNA-polymerase (RdRp), and Maraviroc (MVC), antiretroviral that antagonizes chemokine receptor CCR5, which could affect modulation inflammation viral replication in treatment COVID-19. We sought effect MVC and/or FPV plus systemic steroid (SS) <ns3:italic>vs.</ns3:italic> SS alone on load progression critical disease. Methods Sixteen...

10.12688/f1000research.143776.2 preprint EN cc-by F1000Research 2024-06-11

Introduction: The enzyme lactate dehydrogenase (LDH) is a good marker of general hyperinflammation correlated with mortality for COVID-19, and therefore used in prognosis tools. In current COVID-19 clinical randomized trial (CRT), the blood level LDH was selected as an inclusion criterion. However, decreased during pandemic; hence, impact this decrease on prognostic value evaluated. Methods: Data levels 843 patients were obtained analyzed. Relative risk, standard error receiver operating...

10.2217/bmm-2022-0364 article EN Biomarkers in Medicine 2022-09-02

<ns3:p>Background Coronavirus disease 2019 (COVID-19) has created the need to evaluate drugs such as favipiravir (FPV), an antiviral inhibitor of RNA-dependent RNA-polymerase (RdRp), and Maraviroc (MVC), antiretroviral that antagonizes chemokine receptor CCR5, which could affect modulation inflammation viral replication in treatment COVID-19. We sought effect MVC and/or FPV plus systemic steroid (SS) <ns3:italic>vs.</ns3:italic> SS alone on load progression critical disease. Methods Sixteen...

10.12688/f1000research.143776.1 preprint EN cc-by F1000Research 2024-03-11

In COVID-19 disease, the kidneys are commonly affected; we can see abnormal dipstick or acute kidney injury (AKI), latter of which is associated with mortality in children and adults. NGAL Cystatin C biomarkers that increase after injury. Recognition AKI sometimes late; identification would help to improve outcomes. There few studies evaluating function This work aims describe creatinine urinary levels cystatin C, hematuria, and/or proteinuria patients hospitalized COVID-19.

10.1016/j.ekir.2024.02.432 article EN cc-by-nc-nd Kidney International Reports 2024-04-01

Abstract Multiple studies have established that hyperinflammatory response induced by SARS CoV-2 is a main cause of complications and death in infected subjects. Such dysfunctional immune has been described as dysregulated exacerbated production cytokines chemokines attracts activates inflammatory cells, which start sustain pulmonary systemic damage, thus causing lead to multi organ failure death. Therefore, we suggest blocking key inflammation receptors could help reduce migration...

10.21203/rs.3.rs-107427/v1 preprint EN cc-by Research Square (Research Square) 2020-11-13

Abstract Multiple studies have established that hyperinflammatory response induced by SARS CoV-2 is a main cause of complications and death in infected subjects. Such dysfunctional immune has been described as dysregulated exacerbated production cytokines chemokines attracts activates inflammatory cells, which start sustain pulmonary systemic damage, thus causing lead to multi organ failure death. Therefore, we suggest blocking key inflammation receptors could help reduce migration...

10.21203/rs.3.rs-107427/v4 preprint EN cc-by Research Square (Research Square) 2021-09-10

Abstract Multiple studies have established that hyperinflammatory response induced by SARS CoV-2 is a main cause of complications and death in infected subjects. Such dysfunctional immune has been described as dysregulated exacerbated production cytokines chemokines attracts activates inflammatory cells, which start sustain pulmonary systemic damage, thus causing lead to multi organ failure death. Therefore, we suggest blocking key inflammation receptors could help reduce migration...

10.21203/rs.3.rs-107427/v3 preprint EN cc-by Research Square (Research Square) 2020-11-24

Abstract Multiple studies have established that hyperinflammatory response induced by SARS CoV-2 is a main cause of complications and death in infected subjects. Such dysfunctional immune has been described as dysregulated exacerbated production cytokines chemokines attracts activates inflammatory cells, which start sustain pulmonary systemic damage, thus causing lead to multi organ failure death. Therefore, we suggest blocking key inflammation receptors could help reduce migration...

10.21203/rs.3.rs-107427/v2 preprint EN cc-by Research Square (Research Square) 2020-11-18

Abstract Background : Multiple studies have now established that hyperinflammatory response induced by SARS CoV-2 is a main cause of complications and death in infected subjects. Such dysfunctional immune has been described as dysregulated exacerbated production cytokines chemokines attracts activates inflammatory cells, which start sustain pulmonary systemic damage, thus causing lead to multi organ failure death. Therefore, we suggest blocking key inflammation receptors could help reduce...

10.21203/rs.3.rs-136884/v1 preprint EN cc-by Research Square (Research Square) 2021-01-05
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