M. Kittleson

ORCID: 0000-0003-4492-2691
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About
Contact & Profiles
Research Areas
  • Transplantation: Methods and Outcomes
  • Renal Transplantation Outcomes and Treatments
  • Mechanical Circulatory Support Devices
  • Viral Infections and Immunology Research
  • Cardiac Structural Anomalies and Repair
  • Organ Transplantation Techniques and Outcomes
  • Heart Failure Treatment and Management
  • Organ and Tissue Transplantation Research
  • Cardiovascular Function and Risk Factors
  • Cardiac pacing and defibrillation studies
  • Amyloidosis: Diagnosis, Treatment, Outcomes
  • Pneumocystis jirovecii pneumonia detection and treatment
  • Cytomegalovirus and herpesvirus research
  • Cardiac Valve Diseases and Treatments
  • Cardiovascular Issues in Pregnancy
  • Polyomavirus and related diseases
  • Cardiomyopathy and Myosin Studies
  • Cardiac Arrest and Resuscitation
  • Heparin-Induced Thrombocytopenia and Thrombosis
  • Hepatitis C virus research
  • Diabetes Treatment and Management
  • Congenital Heart Disease Studies
  • Liver Disease and Transplantation
  • Eosinophilic Disorders and Syndromes
  • Potassium and Related Disorders

Cedars-Sinai Medical Center
2016-2025

Cedars-Sinai Smidt Heart Institute
2016-2025

Joint Commission
2024

Saint Luke's Hospital
2024

Clinical Research Institute
2024

Bridgeport Hospital
2023

Sinai Hospital
2012-2023

Association of Chartered Certified Accountants
2020-2022

Cal Humanities
2022

American Heart Association
2021

Importance Clinical guidelines for patients with heart failure reduced ejection fraction (HFrEF) strongly recommend treatment a sodium-glucose cotransporter-2 inhibitor (SGLT2i) to reduce cardiovascular mortality or HF hospitalization. Nationwide adoption of SGLT2i HFrEF in the US is unknown. Objective To characterize patterns use among eligible hospitalized HFrEF. Design, Setting, and Participants This retrospective cohort study analyzed 49 399 across 489 sites Get With The Guidelines–Heart...

10.1001/jamacardio.2023.1266 article EN JAMA Cardiology 2023-05-22

Cardiomyopathy can be initiated by many factors, but the pathways from unique inciting mechanisms to common end point of ventricular dilation and reduced cardiac output are unclear. We previously described a microarray-based prediction algorithm differentiating nonischemic (NICM) ischemic cardiomyopathy (ICM) using nearest shrunken centroids. Accordingly, we tested hypothesis that NICM ICM would have both shared distinct differentially expressed genes relative normal hearts compared gene...

10.1152/physiolgenomics.00255.2004 article EN Physiological Genomics 2005-03-16

Disruption of leptin signaling is associated with obesity, heart failure, and cardiac hypertrophy, but the role in myocyte apoptosis unknown. We tested hypothesis that increases leptin-deficient ob/ob leptin-resistant db/db mice aging left ventricular increased DNA damage, decreased survival. studied young (2- to 3-month-old) old (12- 14-month-old) wild-type (WT) controls (n=2 4 per group). As expected, wall thickness weights were similar among , WT mice, higher versus WT. Young showed...

10.1161/01.res.0000199348.10580.1d article EN Circulation Research 2005-12-09
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