Yuji Takaso

ORCID: 0009-0004-6257-4867
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About
Contact & Profiles
Research Areas
  • Artificial Intelligence in Healthcare and Education
  • Immune Response and Inflammation
  • Neuroinflammation and Neurodegeneration Mechanisms
  • Radiomics and Machine Learning in Medical Imaging
  • Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
  • Radiology practices and education
  • Nerve injury and regeneration
  • Hip and Femur Fractures
  • Frailty in Older Adults
  • Wound Healing and Treatments
  • Cancer Immunotherapy and Biomarkers
  • Hearing, Cochlea, Tinnitus, Genetics
  • Radiopharmaceutical Chemistry and Applications
  • Adenosine and Purinergic Signaling
  • Ion Channels and Receptors
  • Ultrasound in Clinical Applications
  • Calcium signaling and nucleotide metabolism
  • Dementia and Cognitive Impairment Research
  • Facial Nerve Paralysis Treatment and Research
  • COVID-19 diagnosis using AI

Kanazawa University
2012-2024

Jichi Medical University
2024

Fukui Prefectural Hospital
2023

Artificial intelligence models can learn from medical literature and clinical cases generate answers that rival human experts. However, challenges remain in the analysis of complex data containing images diagrams.

10.2196/57054 article EN cc-by JMIR Medical Education 2024-03-09

Abstract Pathological conditions in cochlea, such as ototoxicity, acoustic trauma, and age-related cochlear degeneration, induce cell death the organ of Corti degeneration spiral ganglion neurons (SGNs). Although macrophages play an essential role after injury, its SGNs is limitedly understood. We analyzed status macrophage activation neuronal damage kanamycin-induced unilateral hearing loss mice. The number ionized calcium-binding adapter molecule 1 (Iba1)-positive increased 3 days...

10.1038/s41598-023-43927-9 article EN cc-by Scientific Reports 2023-10-05

Following facial nerve axotomy, function is not fully restored even after reconstruction. This may be attributed to axon degeneration/neuronal death and sustained neuroinflammation. CD38 an enzyme that catalyses the hydrolysis of nicotinamide adenine dinucleotide (NAD+) a candidate molecule for regulating neurodegeneration In this study, we analyzed effect deletion NAD+ supplementation on neuronal glial activation in nucleus brain stem, degeneration immune cell infiltration distal portion...

10.1038/s41598-020-73984-3 article EN cc-by Scientific Reports 2020-10-20

Alzheimer's disease (AD) impairs cognitive functions, subsequently decreasing activity of daily living (ADL), and is frequently accompanied by lower limb fracture including hip in the elderly. However, there have been few studies on what kinds physical functions are affected or degrees dysfunction produced this combination. This study aims to clarify relationship between decreased ADL combination AD fracture.We examined present illness 4340 elderly aged 82.8 ± 9.36 years [average standard...

10.1007/s12199-012-0283-9 article EN cc-by Environmental Health and Preventive Medicine 2012-05-10

<sec> <title>BACKGROUND</title> Artificial intelligence models can learn from medical literature and clinical cases generate answers that rival human experts. However, challenges remain in the analysis of complex data containing images diagrams. </sec> <title>OBJECTIVE</title> This study aims to assess answering capabilities accuracy ChatGPT-4 Vision (GPT-4V) for a set 100 questions, including image-based 2023 otolaryngology board certification examination. <title>METHODS</title> Answers...

10.2196/preprints.57054 preprint EN 2024-02-03

Facial nerve injuries stem from trauma or tumor surgery, triggering neurodegeneration and neuronal cell death in the facial nucleus, consequently inducing irreversible paralysis. Following transection, glial cells are activated undergo proliferation, facilitating motor neuron survival, repair, regeneration. Clinical approaches, including anastomosis hypoglossal grafting, require delicate microscopic techniques. Recent advancements involve reconstruction using polyglycolic acid (PGA) tubes,...

10.7759/cureus.57326 article EN Cureus 2024-03-31

Abstract Immune checkpoint inhibitors can revive exhausted helper T-cells, and inflammatory cell reactivation may cause autoimmune disease–like conditions. Drug-induced arthritis is an immune-related adverse event, but the diagnostic approach undefined. We present utility of 99m Tc-MDP bone scintigraphy for nivolumab-induced arthritis. A 67-year-old man with hypopharyngeal carcinoma presented bilateral multiple metacarpophalangeal joint pain swelling at each nivolumab administration. Regular...

10.1097/rlu.0000000000004861 article EN Clinical Nuclear Medicine 2023-09-23
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