A5067218518- Aluminum toxicity and tolerance in plants and animals
- Fluoride Effects and Removal
- Immune Response and Inflammation
- NF-κB Signaling Pathways
- Folate and B Vitamins Research
- Animal Diversity and Health Studies
- Trace Elements in Health
- Immune cells in cancer
- Curcumin's Biomedical Applications
- Heavy Metal Exposure and Toxicity
- Selenium in Biological Systems
- Bone and Dental Protein Studies
- Immunotoxicology and immune responses
- Probiotics and Fermented Foods
- Cancer, Stress, Anesthesia, and Immune Response
- Cell death mechanisms and regulation
- Macrophage Migration Inhibitory Factor
- Neuroinflammation and Neurodegeneration Mechanisms
- Arsenic contamination and mitigation
- Alzheimer's disease research and treatments
- Biopolymer Synthesis and Applications
- Neuroscience and Neuropharmacology Research
- Milk Quality and Mastitis in Dairy Cows
- Nuclear Receptors and Signaling
- Chemokine receptors and signaling
Shanxi Agricultural University
2023-2024
China Agricultural University
2020-2024
Northeast Agricultural University
2015-2023
Inflammation is the hallmark of extended-spectrum β-lactamase (ESBL)-producing Escherichia coli-induced bovine mastitis. Organic selenium can activate pivotal proteins in immune responses and regulate system. The present study aimed to investigate whether selenomethionine (SeMet) attenuates ESBL E. inflammation mammary epithelial cells (bMECs) macrophages. Cells were treated with 0, 5/10, 10/20, 20/40, or 40/60 μM SeMet for 12 h and/or inoculated ESBL-E. coli [multiplicity infection (MOI) =...
Escherichia coli is a major environmental pathogen causing bovine mastitis, characterized by cell death and mammary tissue damage. Apoptosis, form of death, has an important role in the pathogenesis mastitis. Selenium, essential trace element, protects against mastitis acting through several biochemical pathways, potentially including prevention apoptosis. Our objective was to investigate whether selenomethionine (SeMet) attenuated E. coli-induced apoptosis epithelial cells (bMEC). These...
Fluoride is a pervasive environmental contaminant. Prolonged excessive fluoride intake can inflict severe damage on the liver and intestines. Previous 16S rDNA sequencing revealed decrease in ileal Bifidobacterium abundance during fluoride-induced hepatointestinal injury. Hence, this work aimed to investigate possible mitigating function of injury caused by fluoride. Thirty-six 6-week-old C57BL/6J mice (equally divided between males females) were allotted randomly three groups: Ctrl group...
Fluorosis is a global public health disease affecting more than 50 countries and 500 million people. Excessive fluoride damages the liver intestines, yet mechanisms therapeutic approaches remain unclear.