Adelaide Di Lorenzo

ORCID: 0000-0003-0601-0309
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About
Contact & Profiles
Research Areas
  • Renal and Vascular Pathologies
  • Genetic and Kidney Cyst Diseases
  • Mast cells and histamine
  • Abdominal vascular conditions and treatments
  • Amyloidosis: Diagnosis, Treatment, Outcomes
  • Renal Diseases and Glomerulopathies
  • Apelin-related biomedical research
  • Thyroid Disorders and Treatments
  • Parathyroid Disorders and Treatments
  • Dialysis and Renal Disease Management
  • Pregnancy and preeclampsia studies
  • T-cell and B-cell Immunology
  • Biomarkers in Disease Mechanisms
  • PI3K/AKT/mTOR signaling in cancer
  • Vasculitis and related conditions

University of Foggia
2016-2021

Preeclampsia is a systemic disorder, affecting 2-10% of pregnancies, characterized by deregulated pro- and anti-angiogenic balance. Semaphorin 3F an angiogenesis inhibitor. We aimed to investigate whether semaphorin expression modulated in preeclampsia.We performed two observational single center cohort studies between March 2013 August 2014. In the first we enrolled 110 consecutive women, undergoing elective cesarean section; second included 150 women amniocentesis for routine clinical...

10.1371/journal.pone.0174400 article EN cc-by PLoS ONE 2017-03-28

End-stage renal disease (ESRD) is associated with a broad spectrum of morphological and functional thyroid disorders. Recent studies have shown that low free triiodothyronine (fT3) levels are related to inflammatory status endothelial activation in ESRD patients on haemodialysis (HD). Limited data exist about possible relationship between dialysis regimen, namely long nocturnal (LNHD), function parameters. The aim this study was evaluate the regimen function, consequently main patient...

10.1093/ckj/sfaa003 article EN cc-by-nc Clinical Kidney Journal 2020-01-01

Hypertension is common and occurs in the majority of autosomal dominant polycystic kidney disease (ADPKD) patients prior to loss function. relates progressive enlargement, a significant independent risk factor for progression end-stage renal disease. The pathogenesis hypertension ADPKD complex depends on many factors that influence each other. High expression PKD1 PKD2 genes present cilia tubular epithelial cells, endothelial cells vascular smooth muscle cells. Decreased or absent...

10.1714/1988.21520 article EN PubMed 2015-09-01

Pseudoaneurysm is due to a disruption in arterial wall continuity. It forms sac that communicates with the vessel lumen and surrounded by compressed, surrounding tissues not of artery from which lesion arises. Many causes can predispose formation pseudoaneurysm such as trauma, surgical procedures, anticoagulation. In our patient another important risk factor for ADPKD (autosomal dominant polycystic kidney disease) cause vascular complication. The mechanisms leading genesis pseudoaneurysms...

10.1177/15266028211025035 article EN Journal of Endovascular Therapy 2021-06-17
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