A5015855770- Effects and risks of endocrine disrupting chemicals
- Toxic Organic Pollutants Impact
- Air Quality and Health Impacts
- Carcinogens and Genotoxicity Assessment
- Birth, Development, and Health
- Health, Environment, Cognitive Aging
- Per- and polyfluoroalkyl substances research
- Epigenetics and DNA Methylation
- Heavy Metal Exposure and Toxicity
- Estrogen and related hormone effects
- Microplastics and Plastic Pollution
- Noise Effects and Management
- Nutritional Studies and Diet
- Pesticide Exposure and Toxicity
- Electromagnetic Fields and Biological Effects
- Pregnancy and Medication Impact
- Neonatal Respiratory Health Research
- Sperm and Testicular Function
- Animal testing and alternatives
- Urological Disorders and Treatments
- Pregnancy and preeclampsia studies
- Chemical Analysis and Environmental Impact
- Cancer Risks and Factors
- Climate Change and Health Impacts
- Water Treatment and Disinfection
Instituto de Investigación Biosanitaria de Granada
2016-2025
Universidad de Granada
2016-2025
Centro de Investigación Biomédica en Red de Epidemiología y Salud Pública
2016-2025
Instituto de Salud Carlos III
2014-2025
Universitat de València
2011-2023
Fundación para el Fomento de la Investigación Sanitaria y Biomédica de la Comunitat Valenciana
2015-2023
Barcelona Institute for Global Health
2023
Center for Research in Environmental Epidemiology
2023
Universitat Jaume I
2018-2023
Inserm
2022
Estrogens are defined by their ability to induce the proliferation of cells female genital tract. The wide chemical diversity estrogenic compounds precludes an accurate prediction activity on basis structure. Rodent bioassays not suited for large-scale screening chemicals before release into environment because cost, complexity, and ethical concerns. E-SCREEN assay was developed assess estrogenicity environmental using proliferative effect estrogens target as end point. This quantitative...
Estrogens are defined by their ability to induce the proliferation of cells female genital tract.The wide chemical diversity estrogenic compounds precludes an accurate prediction activity on basis structure.Rodent bioassays not suited for large-scale screening chemicals before release into environment because cost, complexity, and ethical concerns.The E-SCREEN assay was developed assess estrogenicity environmental using proliferative effect estrogens target as end point.This quantitative...
BackgroundExposure to xenoestrogens during pregnancy may disturb the development and function of male sexual organs.ObjectiveIn this study we aimed determine whether combined effect environmental estrogens measured as total effective xenoestrogen burden (TEXB) is a risk factor for urogenital malformations.MethodsIn case–control study, nested in mother–child cohort (n = 702) established at Granada University Hospital, compared 50 newborns with diagnosis cryptorchidism and/or hypospadias 114...
<h3>Background</h3> Air pollution from traffic has been associated with cardiorespiratory diseases in children and adults, but there is little information on its potential neurotoxic effects. This study aimed to investigate the association between exposure nitrogen dioxide (NO<sub>2</sub>), as a marker of traffic-related air pollution, cognitive development children. <h3>Methods</h3> A population-based birth cohort southern Spain was followed age 4 years for 1 year. Complete data analyses...
Prenatal exposure to bisphenol A (BPA) and phthalates may affect fetal growth; however, previous findings are inconsistent based on few studies.We assessed whether prenatal BPA was associated with growth in a Spanish birth cohort of 488 mother-child pairs.We measured eight [four di(2-ethylhexyl) phthalate metabolites (DEHPm), mono-benzyl (MBzP), three low-molecular-weight (LMWPm)] two spot-urine samples collected during the first third trimester pregnancy. We estimated curves for femur...
Author(s): Felix, Janine F; Joubert, Bonnie R; Baccarelli, Andrea A; Sharp, Gemma C; Almqvist, Catarina; Annesi-Maesano, Isabella; Arshad, Hasan; Baiz, Nour; Bakermans-Kranenburg, Marian J; Bakulski, Kelly M; Binder, Elisabeth B; Bouchard, Luigi; Breton, Carrie V; Brunekreef, Bert; Brunst, Burchard, Esteban G; Bustamante, Mariona; Chatzi, Leda; Cheng Munthe-Kaas, Monica; Corpeleijn, Eva; Czamara, Darina; Dabelea, Dana; Davey Smith, George; De Boever, Patrick; Duijts, Liesbeth; Dwyer,...
Breast cancer ranks first in women, and is the second cause of death this gender. In addition to genetics, environment contributes development disease, although factors involved are not well known. Among latter influence microorganisms and, therefore, attention recently being paid mammary microbiota. We hypothesize that risk breast could be associated with composition functionality mammary/gut microbiota, exposure environmental contaminants (endocrine disruptors, EDCs) might contribute alter these
Mitochondria are sensitive to environmental toxicants due their lack of repair capacity. Changes in mitochondrial DNA (mtDNA) content may represent a biologically relevant intermediate outcome mechanisms linking air pollution and fetal growth restriction.We investigated whether placental mtDNA is possible mediator the association between prenatal nitrogen dioxide (NO2) exposure birth weight.We used data from two independent European cohorts: INMA (n = 376; Spain) ENVIRONAGE 550; Belgium)....