Qiu Peng

ORCID: 0000-0001-7085-080X
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About
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Research Areas
  • Ferroptosis and cancer prognosis
  • Intensive Care Unit Cognitive Disorders
  • Autophagy in Disease and Therapy
  • Anesthesia and Neurotoxicity Research
  • Reproductive Biology and Fertility
  • Circular RNAs in diseases
  • Tissue Engineering and Regenerative Medicine
  • Cancer, Lipids, and Metabolism
  • Extracellular vesicles in disease
  • Cancer, Hypoxia, and Metabolism
  • MicroRNA in disease regulation
  • ATP Synthase and ATPases Research
  • 3D Printing in Biomedical Research
  • Metabolism, Diabetes, and Cancer
  • Sperm and Testicular Function
  • Cancer Immunotherapy and Biomarkers
  • Glycosylation and Glycoproteins Research
  • Galectins and Cancer Biology
  • Cancer-related molecular mechanisms research
  • RNA modifications and cancer
  • Traumatic Brain Injury and Neurovascular Disturbances
  • Lipid metabolism and disorders
  • Mycobacterium research and diagnosis
  • Epigenetics and DNA Methylation
  • Mitochondrial Function and Pathology

Hunan Cancer Hospital
2022-2025

Central South University
2022-2025

China Medical University
2023-2024

State Key Laboratory For Conservation and Utilization of Subtropical Agro-Bioresources
2024

Guangxi University
2024

Huazhong University of Science and Technology
2022

Tongji Hospital
2022

Abstract Cellular metabolism is the fundamental process by which cells maintain growth and self‐renewal. It produces energy, furnishes raw materials, intermediates for biomolecule synthesis, modulates enzyme activity to sustain normal cellular functions. foundation of life processes plays a regulatory role in various biological functions, including programmed cell death. Ferroptosis recently discovered form iron‐dependent The inhibition ferroptosis crucial tumorigenesis tumor progression....

10.1002/cac2.12519 article EN cc-by-nc-nd Cancer Communications 2024-01-13

Ferritinophagy, a process involving selective autophagy of ferritin facilitated by nuclear receptor coactivator 4 (NCOA4), entails the recognition NCOA4 and subsequent delivery to autophagosome. Within autophagosome, undergoes degradation, leading release iron in lysosome. It is worth noting that excessive levels can trigger cell death. Recent evidence has elucidated significant roles played ferritinophagy ferroptosis regulation initiation progression cancer. Given crucial role tumor...

10.1038/s41420-023-01753-y article EN cc-by Cell Death Discovery 2023-12-18

Long non‑coding (lnc)RNAs participate in colorectal cancer (CRC) occurrence and progression. The present study aimed to investigate whether lncRNA ABHD11‑AS1 regulates malignant biological behavior of CRC cells. Bioinformatic analysis, reverse transcription‑quantitative PCR situ hybridization revealed that expression was decreased samples associated with an unfavorable prognosis. overexpression significantly proliferation, migration invasion cells, whereas inhibition had the opposite...

10.3892/ijo.2025.5726 article EN cc-by-nc-nd International Journal of Oncology 2025-02-11

Metabolic reprogramming is the survival rule of tumor cells, and cells can meet their high metabolic requirements by changing energy metabolism mode. an important biochemical basis malignant phenotypes. Ras-related C3 botulinum toxin substrate 1 (Rac1) abnormally expressed in a variety tumors plays role proliferation, invasion, migration cells. However, Rac1 still unclear. Herein, we revealed that was highly colon cancer tissues cell lines. promotes migration, invasion upregulating SOX9,...

10.1111/cas.15652 article EN cc-by-nc Cancer Science 2022-11-12

Abstract Background Hepatocellular carcinoma (HCC) is the second major cause of tumor death worldwide, threatening people's lives and health. Circulating cells (CTCs) facilitate progression many cancers, including HCC. Methods Combining transcriptomic data from TCGA, ICGC ctcRbase databases, we characterized differentially expressed circulating cells/microemboli (CTCs/CTMs) related genes (CRGs). These CRGs were then analyzed with a lasso multivariate COX, five-gene prognostic signature was...

10.21203/rs.3.rs-2222900/v1 preprint EN cc-by Research Square (Research Square) 2022-11-02
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