A5072015849- Cardiac Valve Diseases and Treatments
- Coronary Interventions and Diagnostics
- Acute Myocardial Infarction Research
- Cardiac Arrhythmias and Treatments
- Cardiac Structural Anomalies and Repair
- Cardiac Imaging and Diagnostics
- Cardiovascular Function and Risk Factors
- Cardiac electrophysiology and arrhythmias
- Cardiac, Anesthesia and Surgical Outcomes
- Atherosclerosis and Cardiovascular Diseases
- Atrial Fibrillation Management and Outcomes
- Congenital Heart Disease Studies
- Heart Failure Treatment and Management
- Cardiac pacing and defibrillation studies
- Antiplatelet Therapy and Cardiovascular Diseases
- Mechanical Circulatory Support Devices
- Cardiac Health and Mental Health
- Lipoproteins and Cardiovascular Health
- Adipokines, Inflammation, and Metabolic Diseases
- Cardiovascular Disease and Adiposity
- Body Image and Dysmorphia Studies
- Cardiovascular Issues in Pregnancy
- Health and Medical Research Impacts
- Body Contouring and Surgery
- Pulmonary Hypertension Research and Treatments
Instituto Nacional de Cardiología
2015-2024
Universidad César Vallejo
2024
Instituto Nacional de Cardiologia
2001-2022
Hospital del Trabajador
2021
Hospital de São João
2021
Society of Interventional Radiology
2020
Hospital Dipreca
2017
University of Chile
2011-2016
Hospital Clínico de la Universidad de Chile
2014-2016
Interamerican Society of Cardiology
2014
![Graphic][1] Myocardial infarction is a major cause of death and disability worldwide. Coronary atherosclerosis chronic disease with stable unstable periods. During periods activated inflammation in the vascular wall, patients may develop myocardial infarction. Myocardial be minor event lifelong disease, it even go undetected, but also catastrophic leading to sudden or severe haemodynamic deterioration. A first manifestation coronary artery occur, repeatedly, established disease....
Background: The molecular mechanisms underlying acute coronary syndrome (ACS) have been extensively investigated, with a particular focus on the role of circulating microvesicles (MVs) as carriers regulatory elements that influence hemodynamic changes and flow. Endo-thelial platelet dysfunction during ACS alters MVs composition, impacting clinical outcomes. This study explores levels miR–126–5p miR–223–3p in their asso-ciation Thrombolysis Myocardial...
Background. The molecular mechanisms underlying acute coronary syndrome (ACS) have been extensively investigated, with a particular focus on the role of circulating microvesicles (MVs) as carriers regulatory elements that influence hemodynamic changes and flow. Endothelial platelet dysfunction during ACS alters MV composition, impacting clinical outcomes. This study explores levels miR-126-5p miR-223-3p in MVs their association Thrombolysis Myocardial Infarction (TIMI) flow classification...
In large necropsy studies dissecting intramyocardial hematoma (DIH) with serpiginous tracts across the myocardial fibers has been reported in both septum and left ventricle free wall.We studied 15 patients admitted to hospital acute infarction (AMI) which DIH was demonstrated by either transthoracic and/or transesophageal confirmed intraoperatively or necropsy.In nine hemorrhagic dissection predominantly remaining it wall of (LV). Myocardial involved ventricular inferior two, anterior 13...
Background: Inflammation plays an essential role in the development and progression of atherosclerotic lesions plaque disruption. Cytokines are central regulators immunoinflammatory mechanisms. The aim present study was to evaluate INF-γ IL-6 gene polymorphisms as susceptibility markers for acute coronary syndromes (ACS) a group Mexican patients. Methods: Four (INF-γ -155 G>A, -179 T>G, -572 G>C, -1426 T>G) genes were analyzed by 5′ exonuclease TaqMan genotyping assays 284 patients with ACS...
Background: Lipoprotein (Lp(a)) and homocysteine (Hcy) are independent risk factors for coronary artery disease (CAD). Hcy promotes the release of free apo(a) from Lp(a). The high fibrin affinity inhibits plasminogen binding plasmin generation. Hyperhomocysteinemia can result a less active variant methylene tetrahydrofolate reductase (variant C677T). Because C677T genotype is estimated to be present in 32.2% Mexican population, we took advantage this prevalence determine possible...