Diana Argibay
A5091501503- Cancer, Stress, Anesthesia, and Immune Response
- Brain Metastases and Treatment
- Nanoplatforms for cancer theranostics
- Neuroinflammation and Neurodegeneration Mechanisms
- Curcumin's Biomedical Applications
- Cutaneous Melanoma Detection and Management
- Radiomics and Machine Learning in Medical Imaging
- Computational Drug Discovery Methods
- Melanoma and MAPK Pathways
- Epigenetics and DNA Methylation
- Circular RNAs in diseases
- Extracellular vesicles in disease
- MicroRNA in disease regulation
- RNA modifications and cancer
- RNA Interference and Gene Delivery
- Nicotinic Acetylcholine Receptors Study
- vaccines and immunoinformatics approaches
- Ferroptosis and cancer prognosis
- Genomics and Chromatin Dynamics
- CAR-T cell therapy research
- Immunotherapy and Immune Responses
- Advanced biosensing and bioanalysis techniques
- Galectins and Cancer Biology
- Cancer Genomics and Diagnostics
- Cancer Cells and Metastasis
New York University
2017-2023
NYU Langone Health
2020-2022
Eastern Cooperative Oncology Group
2017-2022
Abstract By interacting with proteins and nucleic acids, the vast family of mammalian circRNAs is proposed to influence many biological processes. Here, RNA sequencing analysis differentially expressed during myogenesis revealed that circSamd4 expression increased robustly in mouse C2C12 myoblasts differentiating into myotubes. Moreover, silencing circSamd4, which conserved between human mouse, delayed lowered myogenic markers cultured from both species. Affinity pulldown followed by mass...
Abstract Brain metastasis is a significant cause of morbidity and mortality in multiple cancer types represents an unmet clinical need. The mechanisms that mediate metastatic growth the brain parenchyma are largely unknown. Melanoma, which has highest rate among common types, ideal model to study how cells adapt parenchyma. Our unbiased proteomics analysis melanoma short-term cultures revealed proteins implicated neurodegenerative pathologies differentially expressed explanted from...
Introduction We are conducting a multicenter study to identify classifiers predictive of disease-specific survival in patients with primary melanomas. Here we delineate the unique aspects, challenges, and best practices for optimizing generally small-sized pigmented tumor samples including melanomas at least 1.05mm from AJTCC TNM stage IIA-IIID patients. also evaluated tissue-derived predictors extracted nucleic acids’ quality success downstream testing. This ongoing will target 1,000 within...
Summary Brain metastasis is a significant cause of morbidity and mortality in multiple cancer types represents an unmet clinical need. The mechanisms that mediate metastatic growth the brain parenchyma are largely unknown. Melanoma, which has highest rate among common types, ideal model to study how cells adapt parenchyma. We performed unbiased proteomics analysis melanoma short-term cultures, novel for metastasis. Intriguingly, we found proteins implicated neurodegenerative pathologies...
Patients with stage II and III cutaneous primary melanoma vary considerably in their risk of melanoma-related death. We explore the ability methylation profiling to distinguish classes associations clinicopathologic characteristics survival.
Melanoma is a highly prevalent cancer with an increasing incidence worldwide and high metastatic potential. Brain metastasis major complication of the disease, as more than 50% melanoma patients eventually develop intracranial disease. MicroRNAs (miRNAs) have been found to play important role in tumorigenicity different cancers potential markers disease outcome. Identification relevant miRNAs has generally stemmed from miRNA profiling studies cells or tissues, but these approaches may missed...
<div>Abstract<p>Brain metastasis is a significant cause of morbidity and mortality in multiple cancer types represents an unmet clinical need. The mechanisms that mediate metastatic growth the brain parenchyma are largely unknown. Melanoma, which has highest rate among common types, ideal model to study how cells adapt parenchyma. Our unbiased proteomics analysis melanoma short-term cultures revealed proteins implicated neurodegenerative pathologies differentially expressed...
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
<div>Abstract<p>Brain metastasis is a significant cause of morbidity and mortality in multiple cancer types represents an unmet clinical need. The mechanisms that mediate metastatic growth the brain parenchyma are largely unknown. Melanoma, which has highest rate among common types, ideal model to study how cells adapt parenchyma. Our unbiased proteomics analysis melanoma short-term cultures revealed proteins implicated neurodegenerative pathologies differentially expressed...
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Figure from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis
Supplementary Data from Melanoma-Secreted Amyloid Beta Suppresses Neuroinflammation and Promotes Brain Metastasis