A5103285185- Epilepsy research and treatment
- Pharmacological Effects and Toxicity Studies
- Metabolism and Genetic Disorders
- Amoebic Infections and Treatments
- Parasitic infections in humans and animals
- RNA and protein synthesis mechanisms
- Mosquito-borne diseases and control
- Parasitic Infections and Diagnostics
- Parvovirus B19 Infection Studies
- Complement system in diseases
- Genetics and Neurodevelopmental Disorders
- Virology and Viral Diseases
- Diet and metabolism studies
- Malaria Research and Control
- Ocular Diseases and Behçet’s Syndrome
- Autoimmune Neurological Disorders and Treatments
- Systemic Lupus Erythematosus Research
- Retinal and Optic Conditions
- Respiratory viral infections research
- Infectious Encephalopathies and Encephalitis
- Bacterial Infections and Vaccines
- Chromosomal and Genetic Variations
Krankenhaus Mara
2017
University Hospital Ulm
2017
Universität Ulm
1997-2015
University Medical Center Freiburg
2004
Universitätsklinikum Tübingen
2001
University of Tübingen
2001
ABSTRACT Measles virus (MV) strain CAM/RB, which was adapted to growth in the brain of newborn rodents, is highly neurovirulent. It has been reported earlier that experimentally selected variants escaping from monoclonal antibodies (MAbs) Nc32 and L77 hemagglutinin (H) preserved their neurovirulence, whereas mutants MAbs K71 K29 were found be strongly attenuated (U. G. Liebert et al., J. Virol. 68:1486–1493, 1994). To investigate molecular basis these findings, we have generated a panel...
To compare the frequency of intrathecal immunoglobulin (Ig) synthesis in patients with symptomatic epilepsy and unknown etiology ('cryptogenic').Patients epileptic (n = 301) non-epileptic 10) seizures were retrospectively screened for autochthonous Ig oligoclonal bands (OCBs) cerebrospinal fluid.Intrathecal IgG/OCBs detected 8% epilepsies etiology, 5% first cause 0-4% due to brain tumors, cerebrovascular disease or other etiologies. Intrathecal not seen psychogenic seizures. Identical OCBs...
Einleitung: Dysplastische Veränderungen im temporalen Neokortex werden häufig (bei 10–50%) bei Patienten mit Hippocampussklerose gefunden. Über die Bedeutung dieser für Anfallsentstehung ist bislang wenig bekannt.